Alcohol's Double Effect on Uric Acid
Alcohol raises uric acid through two separate routes, which is why it matters more for gout than almost any food. First, alcohol metabolism competes with uric acid excretion in the kidneys, driving levels up. Second, beer adds a purine load on top — the malt and yeast contribute guanine that wine and distilled spirits largely lack. This double effect explains why beer is the strongest alcohol trigger for gout, spirits sit in the middle, and wine is the least problematic of the three.
Frequently Asked Questions
How many purines are in Sake Lees?
Sake Lees contains about 89 mg of purines per 100g. has moderate purine levels (89.1 mg/100g). For context, purine concentrations above 150 mg/100g are flagged as high-risk in gout care, while under 50 mg is usually considered low-risk.
Can I drink Sake Lees if I have gout?
It is strongly recommended to avoid Sake Lees if you have gout. Alcohol is a well-established gout trigger — it increases uric acid production and reduces its excretion through the kidneys. has moderate purine levels (89.1 mg/100g).The American College of Rheumatology advises gout patients to limit or avoid all types of alcohol, especially during flares.
What food category does Sake Lees belong to?
Sake Lees is classified under Alcoholic Beverages in our gout food database. Its category placement gives helpful framing when comparing foods in your gout diet. Browse other alcoholic beverages items to compare purine and fructose levels across similar foods.
What are gout-friendly alternatives to Sake Lees?
Instead of Sake Lees, consider choosing alcoholic beverages options rated SAFE in our gout database — these have low purine (under 50 mg/100g) and low fructose (under 2g/100g). Browse our alcoholic beverages category page to find the best substitutes for your gout diet plan.
Data Quality
This entry uses T2-level data:
At least one dimension is directly measured; the other is based on scientific consensus.
Sources include USDA FoodData Central (public domain, CC0 1.0), the USDA+ODS-NIH Database for the Purine Content of Foods Release 2.0 (2025), Kaneko et al. Biol Pharm Bull 2014, and ACR/EULAR clinical guidelines.